文献引用
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1) Arteannuin B, a novel NLRP3 inhibitor, suppresses NLRP3 inflammasome in macrophages and alleviates NLRP3-related diseases.
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2) Exenatide through PPARδ improved hepatic insulin resistance in patients of type 2 diabetes mellitus via suppressing pyroptosis.
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3) CX3CR1/P2X4R signaling-mediated spinal microglial M1 polarization contributes to chronic pelvic pain in endometriosis.
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4) Nephropathy 1 Formula (N1F) mitigates cisplatin-induced acute kidney injury by inhibiting TAK1-dependent NF-κB signaling.
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5) Rab32-mediated macrophage apoptosis and apoptotic body release promote M1 polarization in ARDS via the Cxcl11/Ccl4/NF-κB pathway.
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6) Activation of GPR35 in the Anterior Cingulate Cortex Alleviates Neuropathic Pain and Depression-Related Behavior.
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7) Guhan Yangsheng Jing alleviates sleep deprivation-induced neuronal injury via neurotransmitter rebalancing, mitochondrial protection, and inhibition of pyroptosis.
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8) Nitidine chloride alleviates inflammation and pyroptosis by inhibiting the NLRP3/Caspase-1/GSDMD signaling pathway in macrophages and murine models.
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9) Dihuang Yinzi ameliorates post-stroke depression through Miro1 ubiquitination-dependent mitophagy.
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10) HIF-1α drives pyroptosis in extravillous trophoblasts by suppressing PPAR-γ to activate the NLRP3 inflammasome in RA-complicated pregnancy.
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11) Gstk1 confers cardioprotection in sepsis by regulating mitochondrial function and inhibiting cGAS/STING-dependent inflammation and pyroptosis.
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12) Remimazolam mitigates cerebral ischemia/reperfusion injury by concurrently alleviating damage to the blood-brain barrier and RIP3/MLKL-driven necroptosis.
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13) Identifying the active components and mechanisms of Toona sinensis pericarps for the treatment of diabetic kidney disease using network pharmacology and experimental verification.
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14) Jujuboside A ameliorates glomerular podocytes lipotoxicity in diabetic mice by YY1-mediated promotion of intracellular cholesterol transport and efflux.
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15) Subacute ruminal acidosis induces inflammation, autophagy, and apoptosis by disrupting Ca2+ homeostasis via STIM1/ORAI1 upregulation in mammary gland tissues.